A recent research showed that embryonic mouse heart has the capacity to regenerate, a phenomenon that was till now attributed to non-mammalian species. The study was conducted by Professor Timothy C. Cox and his colleagues from the University of Washington in Seattle.
The study showed that the embryonic heart can replace diseased tissue through compensatory proliferation of healthy cells. Disorders of the mitochondria, a cell structure required for energy production, are one of the leading causes of early onset of fatal cardiomyopathies.