According to a new research, scarring from heart attack can be reduced by targeting a protein involved in scar formation. During heart attack, damaged and injured heart gets excessive scar tissues that prevent the heart muscles from working at full capacity.

Researchers at the University of Wisconsin-Madison and Cornell University have identified an important protein, sFRP2 which is instrumental in controlling the formation of excessive tissue or collagen, immediately after a heart attack. Scientist conducted their research on mice and found that the functioning of their heart improved significantly due to the reduced scarring from heart attacks by blocking that specific protein.

One of the researchers, Professor Daniel Greenspan, from the University of Wisconsin-Madison in the US, said, "With many injuries and diseases, large amounts of collagen are formed and deposited in tissues, leading to scarring and a condition called fibrosis." He added, "Importantly, we found that when we reduced the level of fibrosis, heart function significantly improved in mice."

Researchers found that by reducing fibrosis in mice, their heart functioning significantly improved. This activity may provide promising therapies for heart attack damage and improved functioning of impaired heart. Blocking the protein sFRP2 could also help in treatment of other fibrosis conditions including liver cirrhosis and interstitial lung disease. Greenspan said: ""Fibrosis can seriously affect the functioning of heart, lung, liver and other tissues."